Domain analysis reveals striking functional differences between the regulatory subunits of phosphatidylinositol 3-kinase (PI3K), p85α and p85β
Author:
Affiliation:
1. Department of Molecular Medicine, The Scripps Research Institute, La Jolla, San Diego, CA, USA
Publisher
Impact Journals, LLC
Subject
Oncology
Cited by 21 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Unveiling the role of PIK3R1 in cancer: A comprehensive review of regulatory signaling and therapeutic implications;Seminars in Cancer Biology;2024-11
2. PI3K couples long-term synaptic potentiation with cofilin recruitment and actin polymerization in dendritic spines via its regulatory subunit p85α;Cellular and Molecular Life Sciences;2024-08-19
3. A novel TCF3::PIK3R1 fusion linked to decreased PI3K‐AKT signalling activity in paediatric B‐acute lymphoblastic leukaemia;British Journal of Haematology;2024-06-14
4. A p85 isoform switch enhances PI3K activation on endosomes by a MAP4- and PI3P-dependent mechanism;Cell Reports;2024-05
5. Non-catalytic role of phosphoinositide 3-kinase in mesenchymal cell migration through non-canonical induction of p85β/AP2-mediated endocytosis;Nature Communications;2024-03-23
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