WRN helicase defective in the premature aging disorder Werner syndrome genetically interacts with topoisomerase 3 and restores the top3 slow growth phenotype of sgs1 top3-Reference-Cited by-同舟云学术

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WRN helicase defective in the premature aging disorder Werner syndrome genetically interacts with topoisomerase 3 and restores the top3 slow growth phenotype of sgs1 top3

Published:2009-02-05 Issue:2 Volume:1 Page:219-233
ISSN:1945-4589
Container-title:Aging
language:en
Short-container-title:Aging

Author:

Aggarwal Monika¹,Brosh Robert M.¹

Affiliation:

1. Laboratory of Molecular Gerontology, National Institute on Aging, NIH,

Publisher

Impact Journals, LLC

Subject

Cell Biology,Ageing

Cited by 12 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Elucidation of DNA Repair Function of PfBlm and Potentiation of Artemisinin Action by a Small-Molecule Inhibitor of RecQ Helicase;mSphere;2020-12-23

2. Oxidative stress and aging: Learning from yeast lessons;Fungal Biology;2018-06

3. Functional deficit associated with a missense Werner syndrome mutation;DNA Repair;2013-06

4. Functional analyses of human DNA repair proteins important for aging and genomic stability using yeast genetics;DNA Repair;2012-04

5. Introduction and Historical Perspective;Cancer Drug Discovery and Development;2011-08-26

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