A truncated peptide from p35, a Cdk5 activator, prevents Alzheimer's disease phenotypes in model mice-Reference-Cited by-同舟云学术

A truncated peptide from p35, a Cdk5 activator, prevents Alzheimer's disease phenotypes in model mice

Published:2012-10-04 Issue:1 Volume:27 Page:174-186
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Shukla Varsha¹,Zheng Ya‐Li²,Mishra Santosh K.³,Amin Niranjana D.¹,Steiner Joseph⁴,Grant Philip¹,Kesavapany Sashi⁵,Pant Harish C.¹

Affiliation:

1. Laboratory of NeurochemistryNational Institute of Dental and Craniofacial Research, National Institutes of HealthBethesdaMarylandUSA

2. Department of NephrologyNingxia People's HospitalYinchuanChina

3. Molecular Genetics Unit, Laboratory of Sensory BiologyNational Institute of Dental and Craniofacial Research, National Institutes of HealthBethesdaMarylandUSA

4. Neurotherapeutics Development UnitNational Institute of Neurological Disorders and Stroke, National Institute of Dental and Craniofacial Research, National Institutes of HealthBethesdaMarylandUSA

5. Neurobiology Program, Department of BiochemistryYong Loo Lin School of MedicineSingapore

Funder

National Institutes of Health

National Institute of Neurological Disorders and Stroke

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.12-217497

Reference71 articles.

1. Peripherally administered antibodies against amyloid β-peptide enter the central nervous system and reduce pathology in a mouse model of Alzheimer disease