Direct or indirect stimulation of adenosine A 2A receptors enhances bone regeneration as well as bone morphogenetic protein‐2-Reference-Cited by-同舟云学术

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Direct or indirect stimulation of adenosine A 2A receptors enhances bone regeneration as well as bone morphogenetic protein‐2

Published:2015-01-08 Issue:4 Volume:29 Page:1577-1590
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Mediero Aránzazu¹,Wilder Tuere¹,Perez‐Aso Miguel¹,Cronstein Bruce N.¹

Affiliation:

1. Divisions of Translational Medicine and RheumatologyDepartment of MedicineNew York University Langone Medical CenterNew YorkNew YorkUSA

Funder

National Institutes of Health

Takeda Pharmaceuticals North America

Celgene

AstraZeneca

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.14-265066

Reference54 articles.

1. Bone remodeling during fracture repair: The cellular picture

2. Molecular aspects of fracture healing:Which are the important molecules?

3. Trauma-Induced Inflammation and Fracture Healing

4. Bone Morphogenetic Protein-2

5. Use of Recombinant Human Bone Morphogenetic Protein-2 to Achieve Posterolateral Lumbar Spine Fusion in Humans

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1. Translational Experimental Basis of Indirect Adenosine Receptor Agonist Stimulation for Bone Regeneration: A Review;International Journal of Molecular Sciences;2024-06-01

2. Regulatory T cell-derived exosome mediated macrophages polarization for osteogenic differentiation in fracture repair;Journal of Controlled Release;2024-05

3. Three-Dimensional Printing Methods for Bioceramic-Based Scaffold Fabrication for Craniomaxillofacial Bone Tissue Engineering;Journal of Functional Biomaterials;2024-03-01

4. A2A adenosine receptor stimulation ameliorated diabetic-induced osteoporosis in rats;General physiology and biophysics;2024

5. Electroacupuncture stimulating Zusanli (ST36), Sanyinjiao (SP6) in mice with collagen-induced arthritis leads to adenosine A2A receptor-mediated alteration of p38α mitogen-activated protein kinase signaling and inhibition of osteoclastogenesis;J TRADIT CHIN MED;2023

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