Improved exercise capacity in cyclophilin‐D knockout mice associated with enhanced oxygen utilization efficiency and augmented glucose uptake via AMPK‐TBC1D1 signaling nexus-Reference-Cited by-同舟云学术

Improved exercise capacity in cyclophilin‐D knockout mice associated with enhanced oxygen utilization efficiency and augmented glucose uptake via AMPK‐TBC1D1 signaling nexus

Published:2019-07-25 Issue:10 Volume:33 Page:11443-11457
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Radhakrishnan Jeejabai¹²,Baetiong Alvin¹,Kaufman Harrison¹,Huynh Michelle¹,Leschinsky Angela¹,Fresquez Adriana¹³⁴,White Carl⁵³,DiMario Joseph X.⁴⁶,Gazmuri Raúl J.¹²⁷

Affiliation:

1. Resuscitation InstituteRosalind Franklin University of Medicine and ScienceNorth ChicagoIllinoisUSA

2. Department of Clinical SciencesRosalind Franklin University of Medicine and ScienceNorth ChicagoIllinoisUSA

3. Center for Cancer Cell Biology, Immunology, and InfectionRosalind Franklin University of Medicine and ScienceNorth ChicagoIllinoisUSA

4. School of Graduate and Postdoctoral StudiesRosalind Franklin University of Medicine and ScienceNorth ChicagoIllinoisUSA

5. Discipline of Physiology and BiophysicsRosalind Franklin University of Medicine and ScienceNorth ChicagoIllinoisUSA

6. Department of Biomedical ResearchRosalind Franklin University of Medicine and ScienceNorth ChicagoIllinoisUSA

7. Captain James A. Lovell Federal Health Care CenterNorth ChicagoIllinoisUSA

Funder

Rosalind Franklin University of Medicine and Science

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201802238R

Reference76 articles.

1. Cellular functions of immunophilins

2. Cyclophilin-D binds strongly to complexes of the voltage-dependent anion channel and the adenine nucleotide translocase to form the permeability transition pore

3. Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

4. Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death

5. Cyclophilin D controls mitochondrial pore–dependent Ca2+ exchange, metabolic flexibility, and propensity for heart failure in mice

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