Cyclophilin D controls mitochondrial pore–dependent Ca2+ exchange, metabolic flexibility, and propensity for heart failure in mice-Reference-Cited by-同舟云学术

Cyclophilin D controls mitochondrial pore–dependent Ca2+ exchange, metabolic flexibility, and propensity for heart failure in mice

Published:2010-10-01 Issue:10 Volume:120 Page:3680-3687
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Elrod John W.,Wong Renee,Mishra Shikha,Vagnozzi Ronald J.,Sakthievel Bhuvana,Goonasekera Sanjeewa A.,Karch Jason,Gabel Scott,Farber John,Force Thomas,Heller Brown Joan,Murphy Elizabeth,Molkentin Jeffery D.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference46 articles.

1. Mitochondrial Membrane Permeabilization in Cell Death

2. Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death

3. Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

4. Cyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemia

5. Properties of the Permeability Transition Pore in Mitochondria Devoid of Cyclophilin D

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