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Ischemic injury in experimental stroke depends on angiotensin II

Published:2002-02 Issue:2 Volume:16 Page:169-176
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Walther Thomas¹,Olah Laszlo²,Harms Christoph³,Maul Bjoern¹,Bader Michael¹,Hörtnagl Heide³,Schultheiss Heinz-Peter¹,Mies Günter²

Affiliation:

1. Department of Cardiology and PneumologyFree University of BerlinGermany

2. Max-Planck-Institute for Neurological ResearchDepartment of Experimental NeurologyCologneGermany

3. Institute of Pharmacology and ToxicologyMedical Faculty (Charité) of the Humboldt UniversityGermany

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.01-0601com

Reference37 articles.

1. Angiotensin-Converting Enzyme Insertion/Deletion Polymorphism and Cerebrovascular Disease

2. Polymorphism of the angiotensin-converting enzyme (ACE) gene in patients with thrombotic brain infarction

3. Captopril improves neurologic outcome from incomplete cerebral ischemia in rats.

4. Post-stroke treatment with imidapril reduces learning deficits with less formation of brain oedema in a stroke-prone substrain of spontaneously hypertensive rats

5. Blockade of Central Angiotensin AT 1 Receptors Improves Neurological Outcome and Reduces Expression of AP-1 Transcription Factors After Focal Brain Ischemia in Rats

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