Glut2‐dependent glucose‐sensing controls thermoregulation by enhancing the leptin sensitivity of NPY and POMC neurons-Reference-Cited by-同舟云学术

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Glut2‐dependent glucose‐sensing controls thermoregulation by enhancing the leptin sensitivity of NPY and POMC neurons

Published:2010-01-22 Issue:6 Volume:24 Page:1747-1758
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Mounien Lourdes¹,Marty Nell¹,Tarussio David¹,Metref Salima¹,Genoux David¹,Preitner Frédéric¹,Foretz Marc¹,Thorens Bernard¹

Affiliation:

1. Department of Physiology and Center for Integrative Genomics University of Lausanne Lausanne Switzerland

Funder

National Science Foundation

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.09-144923

Reference54 articles.

1. Impaired glucose signaling as a cause of obesity and the metabolic syndrome: the glucoadipostatic hypothesis;Mobbs C. V.;Physiol. Behav.,2004

2. Brain Glucose Sensing, Counterregulation, and Energy Homeostasis

3. Activity of single neurons in the hypothalamic feeding centers: effect of glucose

4. Convergence of hepatoportal glucose-sensitive afferent signals to glucose-sensitive units within the nucleus of the solitary tract

5. Glucose-sensing neurons

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