Ablation of the very‐long‐chain fatty acid elongase ELOVL3 in mice leads to constrained lipid storage and resistance to diet‐induced obesity-Reference-Cited by-同舟云学术

Ablation of the very‐long‐chain fatty acid elongase ELOVL3 in mice leads to constrained lipid storage and resistance to diet‐induced obesity

Published:2010-07-06 Issue:11 Volume:24 Page:4366-4377
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Zach‐Avec Damir¹,Brolinson Annelie¹,Fisher Rachel M.²,Carneheim Claes¹,Csikasz Robert I.¹,Bertrand‐Michel Justine³,Borén Jan⁴,Guillou Hervé⁵,Rudling Mats⁶,Jacobsson Anders¹

Affiliation:

1. Wenner‐Gren InstituteLaboratories F3Stockholm University Stockholm Sweden

2. Atherosclerosis Research UnitDepartment of Medicine (Solna)Karolinska InstitutetKarolinska University Hospital Stockholm Sweden

3. IFR150Institut Fédératif de Recherche Bio‐Médicale de ToulouseCHU Purpan Toulouse France

4. Sahlgrenska Center for Cardiovascular and Metabolic ResearchWallenberg LaboratorySahlgrenska University Hospital Göteborg Sweden

5. Laboratoire de Pharmacologie et ToxicologieInstitut National de la Recherche Agronomique (INRA) UR66 Toulouse France

6. Department of Endocrinology, Metabolism and DiabetesMolecular Nutrition UnitKarolinska InstitutetKarolinska University Hospital Huddinge Stockholm Sweden

Funder

Vetenskapsrådet

Institut National de la Recherche Agronomique

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.09-152298

Reference44 articles.

1. Deletion of ELOVL5 leads to fatty liver through activation of SREBP-1c in mice

2. Crucial role of a long-chain fatty acid elongase, Elovl6, in obesity-induced insulin resistance