Unacylated ghrelin normalizes skeletal muscle oxidative stress and prevents muscle catabolism by enhancing tissue mitophagy in experimental chronic kidney disease-Reference-Cited by-同舟云学术

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Unacylated ghrelin normalizes skeletal muscle oxidative stress and prevents muscle catabolism by enhancing tissue mitophagy in experimental chronic kidney disease

Published:2017-08-04 Issue:12 Volume:31 Page:5159-5171
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Gortan Cappellari Gianluca¹,Semolic Annamaria¹,Ruozi Giulia²,Vinci Pierandrea¹,Guarnieri Gianfranco¹,Bortolotti Francesca²,Barbetta Davide³,Zanetti Michela¹,Giacca Mauro²,Barazzoni Rocco¹

Affiliation:

1. Department of Medical Surgical, and Health SciencesUniversity of Trieste Trieste Italy

2. Molecular Medicine LaboratoryInternational Centre for Genetic Engineering and Biotechnology Trieste Italy

3. Animal FacilityUniversity of Trieste Trieste Italy

Funder

Università degli Studi di Trieste

International Centre for Genetic Engineering and Biotechnology

European Society for Clinical Nutrition and Metabolism

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201700126R

Reference50 articles.

1. Indoxyl sulfate potentiates skeletal muscle atrophy by inducing the oxidative stress-mediated expression of myostatin and atrogin-1

2. Mechanisms of muscle wasting in chronic kidney disease

3. Markers of oxidative stress in the skeletal muscle of patients on haemodialysis

4. Insulin resistance in chronic kidney disease: new lessons from experimental models

5. Strong association between malnutrition, inflammation, and atherosclerosis in chronic renal failure

Cited by 36 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Unacylated Ghrelin Protects Against Age‐Related Loss of Muscle Mass and Contractile Dysfunction in Skeletal Muscle;Aging Cell;2024-09-02

2. Mitochondrial targeted catalase improves muscle strength following arteriovenous fistula creation in mice with chronic kidney disease;Scientific Reports;2024-04-09

3. Common mouse models of chronic kidney disease are not associated with cachexia;Communications Biology;2024-03-20

4. Mitochondrial dysfunction and mitophagy blockade contribute to renal osteodystrophy in chronic kidney disease-mineral bone disorder;2023-12-26

5. N-acetylcysteine treatment attenuates hemodialysis access-related limb pathophysiology in mice with chronic kidney disease;American Journal of Physiology-Renal Physiology;2023-09-01

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