EGCG ameliorates high‐fat– and high‐fructose‐induced cognitive defects by regulating the IRS/AKT and ERK/CREB/BDNF signaling pathways in the CNS-Reference-Cited by-同舟云学术

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EGCG ameliorates high‐fat– and high‐fructose‐induced cognitive defects by regulating the IRS/AKT and ERK/CREB/BDNF signaling pathways in the CNS

Published:2017-07-31 Issue:11 Volume:31 Page:4998-5011
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Mi Yashi¹,Qi Guoyuan¹,Fan Rong¹,Qiao Qinglian¹,Sun Yali¹,Gao Yuqi¹,Liu Xuebo¹

Affiliation:

1. Laboratory of Functional Chemistry and Nutrition of FoodCollege of Food Science and EngineeringNorthwest A&F UniversityYanglingChina

Funder

National Natural Science Foundation of China

National Basic Research Program of China

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201700400RR

Reference67 articles.

1. High fat diet produces brain insulin resistance, synaptodendritic abnormalities and altered behavior in mice

2. High-Fat Diet Induces Hepatic Insulin Resistance and Impairment of Synaptic Plasticity

3. Insulin Resistance and Alzheimer-like Reductions in Regional Cerebral Glucose Metabolism for Cognitively Normal Adults With Prediabetes or Early Type 2 Diabetes

4. Insulin and neurodegenerative disease: shared and specific mechanisms

5. Sesamol ameliorates high-fat and high-fructose induced cognitive defects via improving insulin signaling disruption in the central nervous system

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