KR‐31378 ameliorates atherosclerosis by blocking monocyte recruitment in hypercholestrolemic mice

Author:

Kim Jiyun1,Nam Ki‐Hoan1,Kim Sun‐Ok2,Choi Jae‐Hoon1,Kim Hyoung‐Chin3,Yang Sung‐Don4,Kang Joo‐Hyoung1,Ryu Young‐Han1,Taeg Oh Goo1,Yoo Sung‐Eun4

Affiliation:

1. Laboratory of Development and DifferentiationKorea Research Institute of Bioscience and BiotechnologyYusongDaejon305‐333

2. AgroPharma Research InstituteDongbu Hannong Chemical Co. Ltd.Daeduck Science TownDaejon305‐708

3. Laboratory of Experimental Animal ResourcesKorea Research Institute of Bioscience and BiotechnologyYusongDaejon305‐333

4. Center for Biological Modulators of the 21st Century Frontier ProgramKorea Research Institute of Chemical TechnologyYusongDaejon305‐343Korea

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Reference54 articles.

1. The pathogenesis of atherosclerosis: a perspective for the 1990s

2. Chemokines and atherosclerosis

3. Tumor necrosis factor signaling

4. NF-κB: pivotal mediator or innocent bystander in atherogenesis?

5. Inhibition of nuclear factor‐kappa B nuclear localization reduces human E‐selectin expression and the systemic inflammatory response;Boyle E. M.;Circulation,1998

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