KR-31543 reduces the production of proinflammatory molecules in human endothelial cells and monocytes and attenuates atherosclerosis in mouse model-Reference-Cited by-同舟云学术

KR-31543 reduces the production of proinflammatory molecules in human endothelial cells and monocytes and attenuates atherosclerosis in mouse model

Published:2012 Issue:12 Volume:44 Page:733
ISSN:1226-3613
Container-title:Experimental & Molecular Medicine
language:en
Short-container-title:Exp Mol Med

Author:

Choi Jae-Hoon¹,Yoo Ji-Young²,Kim Sun-Ok³,Yoo Sung-Eun⁴,Oh Goo Taeg²

Affiliation:

1. Laboratory of Molecular Physiology, Department of Life Science, College of Natural Sciences, Research Institute for Natural Sciences, Hanyang University, Seoul 133-791, Korea.

2. Division of Life and Pharmaceutical Sciences, Ewha Womans University, Seoul 120-750, Korea.

3. Samyang Corporation, Daejeon 305-717, Korea.

4. Graduate School of New Drug Discovery and Development, Chungnam National University, Daejon 305-764, Korea.

Publisher

Springer Science and Business Media LLC

Subject

Clinical Biochemistry,Molecular Biology,Molecular Medicine,Biochemistry

Reference25 articles.

1. Potential Antiatherogenic Mechanisms of Ascorbate (Vitamin C) and α-Tocopherol (Vitamin E)

2. Insulin-Like Growth Factor-1 Enhances Inflammatory Responses in Endothelial Cells

3. Hematein Inhibits Atherosclerosis by Inhibition of Reactive Oxygen Generation and NF-κB–Dependent Inflammatory Mediators in Hyperlipidemic Mice

4. Oxidized low-density lipoprotein induces the production of interleukin-8 by endothelial cells

5. NF-κB: pivotal mediator or innocent bystander in atherogenesis?

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