Openers of SK Ca and IK Ca channels enhance agonist‐evoked endothelial nitric oxide synthesis and arteriolar vasodilation-Reference-Cited by-同舟云学术

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Openers of SK Ca and IK Ca channels enhance agonist‐evoked endothelial nitric oxide synthesis and arteriolar vasodilation

Published:2008-12-12 Issue:4 Volume:23 Page:1138-1145
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Sheng Jian-zhong¹,Ella Srikanth²,Davis Michael J.²,Hill Michael A.²,Braun Andrew P.¹

Affiliation:

1. Department of Pharmacology and Therapeutics Smooth Muscle Research GroupLibin Cardiovascular InstituteUniversity of Calgary Calgary Alberta Canada

2. Department of Medical Pharmacology and Physiology Dalton Cardiovascular Research CenterUniversity of Missouri Columbia Missouri USA

Funder

Canadian Institutes of Health Research

Alberta Heritage Foundation for Medical Research

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.08-120451

Reference33 articles.

1. Combination of Ca2+ -activated K+ channel blockers inhibits acetylcholine-evoked nitric oxide release in rat superior mesenteric artery

2. Small- and intermediate-conductance Ca2+-activated K+ channels directly control agonist-evoked nitric oxide synthesis in human vascular endothelial cells

3. EDHF: bringing the concepts together

4. Small‐ and Intermediate‐Conductance Calcium‐Activated K + Channels Provide Different Facets of Endothelium‐Dependent Hyperpolarization in Rat Mesenteric Artery

5. Evidence for Involvement of Both IK Ca and SK Ca Channels in Hyperpolarizing Responses of the Rat Middle Cerebral Artery

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