Ischemia‐reperfusion selectively impairs nitric oxide‐ mediated dilation in coronary arterioles: counteracting role of arginase-Reference-Cited by-同舟云学术

Ischemia‐reperfusion selectively impairs nitric oxide‐ mediated dilation in coronary arterioles: counteracting role of arginase

Published:2003-10-16 Issue:15 Volume:17 Page:2328-2330
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Hein Travis W.¹,Zhang Cuihua¹,Wang Wei¹,Chang Chiung-I¹,Thengchaisri Naris¹,Kuo Lih¹

Affiliation:

1. Department of Medical Physiology, Cardiovascular Research Institute, College of MedicineTexas A&M University System Health Science CenterCollege StationTexas

Funder

National Heart, Lung, and Blood Institute

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.03-0115fje

Reference49 articles.

1. Shifting the open-artery hypothesis downstream: the quest for optimal reperfusion

2. Lack of myocardial perfusion immediately after successful thrombolysis. A predictor of poor recovery of left ventricular function in anterior myocardial infarction.

3. Myocardial Perfusion Patterns Related to Thrombolysis in Myocardial Infarction Perfusion Grades After Coronary Angioplasty in Patients With Acute Anterior Wall Myocardial Infarction

4. Pathophysiology of ischaemia-reperfusion injury

5. Coronary Vascular Reactivity After Acute Myocardial Ischemia

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