Overexpression of UCP3 in cultured human muscle lowers mitochondrial membrane potential, raises ATP/ADP ratio, and favors fatty acid versus glucose oxidation-Reference-Cited by-同舟云学术

Overexpression of UCP3 in cultured human muscle lowers mitochondrial membrane potential, raises ATP/ADP ratio, and favors fatty acid versus glucose oxidation

Published:2001-07-24 Issue:11 Volume:15 Page:2033-2035
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

García‐Martínez Cèlia¹,Sibille Brigitte²,Solanes Gemma¹,Darimont Christian³,Macé Katherine³,Villarroya Francesc¹,Gómez‐Foix Anna M.

Affiliation:

1. Departament de Bioquímica i Biología MolecularUniversitat de BarcelonaBarcelona08028Spain

2. Laboratoire de Physiologie et Regulations EnergétiquesCellulaires et MoleculairesUniversité Claude Bernard‐Lyonl43, bld 11 Novembre 1918Villeurbanne69622France

3. Nestlé Research CenterLausanneSwitzerland

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.00-0828fje

Reference38 articles.

1. UCP3: An Uncoupling Protein Homologue Expressed Preferentially and Abundantly in Skeletal Muscle and Brown Adipose Tissue

2. Energy metabolism in uncoupling protein 3 gene knockout mice;Vidal‐Puig A. J.;J. Biol. Chem.,2000

3. Lack of Obesity and Normal Response to Fasting and Thyroid Hormone in Mice Lacking Uncoupling Protein-3

4. Mice overexpressing human uncoupling protein-3 in skeletal muscle are hyperphagic and lean

5. Effects of mutations in the human uncoupling protein 3 gene on the respiratory quotient and fat oxidation in severe obesity and type 2 diabetes.

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