Leydig cell steroidogenesis unexpectedly escapes mitochondrial dysfunction in prematurely aging mice-Reference-Cited by-同舟云学术

Leydig cell steroidogenesis unexpectedly escapes mitochondrial dysfunction in prematurely aging mice

Published:2015-08 Issue:8 Volume:29 Page:3274-3286
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:The FASEB Journal

Author:

Shabalina Irina G.,Landreh Luise,Edgar Daniel,Hou Mi,Gibanova Natalia,Atanassova Nina,Petrovic Natasa,Hultenby Kjell,Söder Olle,Nedergaard Jan,Svechnikov Konstantin

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Reference39 articles.

1. Premature ageing in mice expressing defective mitochondrial DNA polymerase

2. Mitochondrial DNA Mutations, Oxidative Stress, and Apoptosis in Mammalian Aging

3. Mitochondrial DNA Mutations Induce Mitochondrial Dysfunction, Apoptosis and Sarcopenia in Skeletal Muscle of Mitochondrial DNA Mutator Mice

4. Random Point Mutations with Major Effects on Protein-Coding Genes Are the Driving Force behind Premature Aging in mtDNA Mutator Mice

5. Energized, Polarized, and Actively Respiring Mitochondria Are Required for Acute Leydig Cell Steroidogenesis

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