Differential responses to UCP1 ablation in classical brown versus beige fat, despite a parallel increase in sympathetic innervation
Author:
Funder
China Scholarship Council
Novo Nordisk Foundation
Diabetesfonden
Magnus Bergvalls Stiftelse
Swedish Research Council
Carl Tryggers Stiftelse for Vetenskaplig Forskning
Publisher
Elsevier BV
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1. Mice lacking mitochondrial uncoupling protein are cold-sensitive but not obese;Enerback;Nature,1997
2. Only UCP1 can mediate adaptive nonshivering thermogenesis in the cold;Golozoubova;FASEB J.,2001
3. UCP1 deficiency causes brown fat respiratory chain depletion and sensitizes mitochondria to calcium overload-induced dysfunction;Kazak;Proc. Natl. Acad. Sci. U. S. A.,2017
4. Long-term cold Adaptation does not require FGF21 or UCP1;Keipert;Cell Metab.,2017
5. Loss of UCP1 function augments recruitment of futile lipid cycling for thermogenesis in murine brown fat;Oeckl;Mol. Metab.,2022
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