β1 integrin monoclonal antibody treatment ameliorates cerebral cavernous malformations-Reference-Cited by-同舟云学术

β1 integrin monoclonal antibody treatment ameliorates cerebral cavernous malformations

Published:2022-11-09 Issue:12 Volume:36 Page:
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:The FASEB Journal

Author:

McCurdy Sara¹^ORCID,Lin Jenny¹,Shenkar Robert²,Moore Thomas²,Lightle Rhonda²,Faurobert Eva³^ORCID,Lopez‐Ramirez Miguel‐Alejandro¹⁴^ORCID,Awad Issam²,Ginsberg Mark H.¹^ORCID

Affiliation:

1. Department of Medicine University of California San Diego La Jolla California USA

2. Department of Neurological Surgery University of Chicago Chicago Illinois USA

3. Institute for Advanced Biosciences, Inserm 1209, CNRS 5309 Université de Grenoble Alpes Grenoble France

4. Department of Pharmacology University of California, San Diego La Jolla California USA

Funder

National Institute of Neurological Disorders and Stroke

NHLBI Division of Intramural Research

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.202200907RR

Reference38 articles.

1. EndMT contributes to the onset and progression of cerebral cavernous malformations

2. Endothelial Expression of β1 Integrin Is Required for Embryonic Vascular Patterning and Postnatal Vascular Remodeling

3. β1 integrin expression on endothelial cells is required for angiogenesis but not for vasculogenesis

4. Cell-autonomous requirement for β1 integrin in endothelial cell adhesion, migration and survival during angiogenesis in mice

5. Integrin β1 controls VE-cadherin localization and blood vessel stability

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