EndMT contributes to the onset and progression of cerebral cavernous malformations-Reference-Cited by-同舟云学术

EndMT contributes to the onset and progression of cerebral cavernous malformations

Published:2013-06 Issue:7455 Volume:498 Page:492-496
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Maddaluno Luigi,Rudini Noemi,Cuttano Roberto,Bravi Luca,Giampietro Costanza,Corada Monica,Ferrarini Luca,Orsenigo Fabrizio,Papa Eleanna,Boulday Gwenola,Tournier-Lasserve Elisabeth,Chapon Françoise,Richichi Cristina,Retta Saverio Francesco,Lampugnani Maria Grazia,Dejana Elisabetta

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature12207.pdf

Reference37 articles.

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2. Whitehead, K. J. et al. The cerebral cavernous malformation signaling pathway promotes vascular integrity via Rho GTPases. Nature Med. 15, 177–184 (2009)

3. Chan, A. C. et al. Mutations in 2 distinct genetic pathways result in cerebral cavernous malformations in mice. J. Clin. Invest. 121, 1871–1881 (2011)

4. Glading, A., Han, J., Stockton, R. A. & Ginsberg, M. H. KRIT-1/CCM1 is a Rap1 effector that regulates endothelial cell cell junctions. J. Cell Biol. 179, 247–254 (2007)

5. Stockton, R. A., Shenkar, R., Awad, I. A. & Ginsberg, M. H. Cerebral cavernous malformations proteins inhibit Rho kinase to stabilize vascular integrity. J. Exp. Med. 207, 881–896 (2010)

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