Prostanoid receptors in hyperfiltration‐mediated glomerular injury: Novel agonists and antagonists reveal opposing roles for <scp>EP2</scp> and <scp>EP4</scp> receptors-Reference-Cited by-同舟云学术

Prostanoid receptors in hyperfiltration‐mediated glomerular injury: Novel agonists and antagonists reveal opposing roles for EP2 and EP4 receptors

Published:2022-09-20 Issue:10 Volume:36 Page:
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:The FASEB Journal

Author:

Srivastava Tarak¹²³^ORCID,Garola Robert E.⁴,Zhou Jianping²⁵,Boinpelly Varun C.²⁵,Priya Lakshmi¹,Ali Mohammed Farhan¹,Rezaiekhaligh Mohammad H.¹,Heruth Daniel P.⁶,Novak Jan⁷,Alon Uri S.¹,Joshi Trupti⁸⁹¹⁰¹¹,Jiang Yuexu⁹¹⁰,McCarthy Ellen T.¹²,Savin Virginia J.⁵¹²,Johnson Mark L.³,Sharma Ram⁵,Sharma Mukut²⁵¹²

Affiliation:

1. Section of Nephrology, Children's Mercy Hospital and University of Missouri at Kansas City Kansas City Missouri USA

2. Midwest Veterans' Biomedical Research Foundation (MVBRF) Kansas City Missouri USA

3. Department of Oral and Craniofacial Sciences University of Missouri at Kansas City—School of Dentistry Kansas City Missouri USA

4. Department of Pathology and Laboratory Medicine Children's Mercy Hospital and University of Missouri at Kansas City Kansas City Missouri USA

5. Renal Research Laboratory Kansas City VA Medical Center Kansas City Missouri USA

6. Children's Mercy Research Institute Children's Mercy Hospital and University of Missouri at Kansas City Kansas City Missouri USA

7. Department of Microbiology University of Alabama at Birmingham Birmingham Alabama USA

8. Department of Health Management and Informatics University of Missouri Columbia Missouri USA

9. Department of Electrical Engineering and Computer Science University of Missouri Columbia Missouri USA

10. Christopher S. Bond Life Sciences Center University of Missouri Columbia Missouri USA

11. MU Institute for Data Science and Informatics University of Missouri Columbia Missouri USA

12. Department of Internal Medicine, The Jared Grantham Kidney Institute University of Kansas Medical Center Kansas City Kansas USA

Funder

Division of Diabetes, Endocrinology, and Metabolic Diseases

U.S. Department of Veterans Affairs

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.202200875R

Reference95 articles.

1. A Potential Role for Mechanical Forces in the Detachment of Podocytes and the Progression of CKD

2. Potential relevance of shear stress for slit diaphragm and podocyte function

3. Hyperfiltration-associated biomechanical forces in glomerular injury and response: Potential role for eicosanoids

4. Role of biomechanical forces in hyperfiltration-mediated glomerular injury in congenital anomalies of the kidney and urinary tract

5. Podocytes are sensitive to fluid shear stress in vitro

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1. Tissue lipidomic profiling supports a mechanistic role of the prostaglandin E2 pathway for albuminuria development in glomerular hyperfiltration;Frontiers in Network Physiology;2023-12-22

2. Studies in Zebrafish and Rat Models Support Dual Blockade of EP2 and EP4 (Prostaglandin E ₂ Receptors Type 2 and 4) for Renoprotection in Glomerular Hyperfiltration and Albuminuria;Hypertension;2023-04