A cryptic non–GPI‐anchored cytosolic isoform of CD59 controls insulin exocytosis in pancreatic β‐cells by interaction with SNARE proteins-Reference-Cited by-同舟云学术

A cryptic non–GPI‐anchored cytosolic isoform of CD59 controls insulin exocytosis in pancreatic β‐cells by interaction with SNARE proteins

Published:2019-08-14 Issue:11 Volume:33 Page:12425-12434
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Golec Ewelina¹,Rosberg Rebecca¹,Zhang Enming²,Renström Erik²,Blom Anna M.¹,King Ben C.¹

Affiliation:

1. Section of Medical Protein Chemistry, Department of Translational MedicineLund UniversityMalmöSweden

2. Department of Clinical Sciences MalmöLund University Diabetes CentreMalmöSweden

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201901007R

Reference25 articles.

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3. Glycation of the Complement Regulatory Protein CD59 Is a Novel Biomarker for Glucose Handling in Humans

4. Intracellular complement − the complosome − in immune cell regulation

5. Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation

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