Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation-Reference-Cited by-同舟云学术

Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation

Published:2019-01 Issue:1 Volume:29 Page:202-210.e6
ISSN:1550-4131
Container-title:Cell Metabolism
language:en
Short-container-title:Cell Metabolism

Author:

King Ben C.,Kulak Klaudia,Krus Ulrika,Rosberg Rebecca,Golec Ewelina,Wozniak Katarzyna,Gomez Maria F.,Zhang Enming,O'Connell David J.,Renström Erik,Blom Anna M.^ORCID

Funder

Swedish Research Council

Knut and Alice Wallenberg Foundation

Diabetesfonden

Greta and Johan Kocks Foundation

Royal Physiographical Society in Lund

Lars Hiertas Memorial fund

Albert Påhlsson fund

Crafoord Foundation

Tore Nilssons Foundation for Medical Research

Swedish Heart-Lung Foundation

Swedish Foundation for Strategic Research

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

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