Evidence of vasculature and chondrocyte to osteoblast transdifferentiation in craniofacial synovial joints: Implications for osteoarthritis diagnosis and therapy-Reference-Cited by-同舟云学术

Evidence of vasculature and chondrocyte to osteoblast transdifferentiation in craniofacial synovial joints: Implications for osteoarthritis diagnosis and therapy

Published:2020-02-06 Issue:3 Volume:34 Page:4445-4461
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Ruscitto Angela¹,Morel Mallory M.¹,Shawber Carrie J.²,Reeve Gwendolyn³,Lecholop Michael K.⁴,Bonthius Daniel⁵,Yao Hai⁵⁶,Embree Mildred C.¹

Affiliation:

1. Cartilage Biology and Regenerative Medicine Laboratory, College of Dental Medicine Columbia University Irving Medical Center New York NY USA

2. Department of OB/GYN, Division of Reproductive Sciences, College of Physicians and Surgeons Columbia University Irving Medical Center New York NY USA

3. Division of Oral and Maxillofacial Surgery New York Presbyterian Weill Cornell Medical Center New York NY USA

4. Department of Oral and Maxillofacial Surgery, College of Dental Medicine Medical University of South Carolina Charleston SC USA

5. Clemson‐MUSC Bioengineering Program, Department of Bioengineering Clemson University Greenville SC USA

6. Department of Oral Health Sciences Medical University of South Carolina Charleston SC USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201902287R

Reference96 articles.

1. Phenotypic instability of chondrocytes in osteoarthritis: on a path to hypertrophy

2. Radiocarbon dating reveals minimal collagen turnover in both healthy and osteoarthritic human cartilage

3. Biglycan and Fibromodulin Have Essential Roles in Regulating Chondrogenesis and Extracellular Matrix Turnover in Temporomandibular Joint Osteoarthritis

4. Accelerated osteoarthritis in the temporomandibular joint of biglycan/fibromodulin double-deficient mice

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