Intracellular acidosis‐activated p38 MAPK signaling and its essential role in cardiomyocyte hypoxic injury-Reference-Cited by-同舟云学术

Intracellular acidosis‐activated p38 MAPK signaling and its essential role in cardiomyocyte hypoxic injury

Published:2004-10-15 Issue:1 Volume:19 Page:109-111
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Zheng Ming¹,Reynolds Cherilynn²,Jo Su‐Hyun²,Wersto Robert³,Han Qide¹,Xiao Rui‐Ping¹²

Affiliation:

1. Institute of Molecular MedicineInstitute of Cardiovascular Sciences, and Key Laboratory of Molecular Cardiovascular Biology of Chinese Ministry of Education, Peking UniversityBeijing100871People's Republic of China

2. Laboratory of Cardiovascular ScienceGerontology Research Center, National Institute on Aging, NIHBaltimoreMD21224

3. Flow Cytometry LaboratoryGerontology Research Center, National Institute on Aging, NIHBaltimoreMD21224

Funder

National Natural Science Foundation of China

National Institutes of Health

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.04-2607fje

Reference43 articles.

1. Intracellular Acidification Triggered by Mitochondrial-derived Hydrogen Peroxide Is an Effector Mechanism for Drug-induced Apoptosis in Tumor Cells

2. Apoptosis Induced by IL-2 Withdrawal Is Associated with an Intracellular Acidification

3. Apoptosis induced in Jurkat cells by several agents is preceded by intracellular acidification.

4. Effect of 5-minute ischemia on regional pH and energy state of the gerbil brain: relation to selective vulnerability of the hippocampus.

5. Preconditioning rabbit cardiomyocytes: role of pH, vacuolar proton ATPase, and apoptosis.

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