Slack K + channels attenuate NMDA‐induced excitotoxic brain damage and neuronal cell death-Reference-Cited by-同舟云学术

Slack K + channels attenuate NMDA‐induced excitotoxic brain damage and neuronal cell death

Published:2021-04-05 Issue:5 Volume:35 Page:
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Ehinger Rebekka¹,Kuret Anna¹,Matt Lucas¹,Frank Nadine¹,Wild Katharina¹,Kabagema‐Bilan Clement¹,Bischof Helmut¹,Malli Roland²,Ruth Peter¹,Bausch Anne E.¹,Lukowski Robert¹

Affiliation:

1. Department of Pharmacology Toxicology and Clinical Pharmacy Institute of Pharmacy University of Tübingen Tübingen Germany

2. Gottfried Schatz Research Center, Molecular Biology and Biochemistry Medical University of Graz Graz Austria

Funder

Deutsche Forschungsgemeinschaft

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.202002308RR

Reference104 articles.

1. Formation of intermediate-conductance calcium-activated potassium channels by interaction of Slack and Slo subunits

2. Localization of the Slack potassium channel in the rat central nervous system

3. For K channels, Na is the new Ca

4. Amino-termini isoforms of the Slack K+channel, regulated by alternative promoters, differentially modulate rhythmic firing and adaptation

5. Differential distribution of the sodium-activated potassium channels slick and slack in mouse brain

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