Differential distribution of the sodium-activated potassium channels slick and slack in mouse brain
Author:
Affiliation:
1. Division of Molecular and Cellular Pharmacology; Medical University of Innsbruck; 6020 Innsbruck Austria
2. Department of Pharmacology; Medical University of Innsbruck; 6020 Innsbruck Austria
Funder
Austrian Research Fund PhD program
Publisher
Wiley
Subject
General Neuroscience
Reference43 articles.
1. De novo gain-of-function KCNT1 channel mutations cause malignant migrating partial seizures of infancy;Barcia;Nat Genet,2012
2. TrpC3/C7 and Slo2.1 are molecular targets for metabotropic glutamate receptor signaling in rat striatal cholinergic interneurons;Berg;J Neurosci,2007
3. For K+ channels, Na+ is the new Ca2+;Bhattacharjee;Trends Neurosci,2005
4. Localization of the Slack potassium channel in the rat central nervous system;Bhattacharjee;J Comp Neurol,2002
5. Localization of the Na+-activated K+ channel Slick in the rat central nervous system;Bhattacharjee;J Comp Neurol,2005
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