sPLA2‐IB and PLA2R mediate insufficient autophagy and contribute to podocyte injury in idiopathic membranous nephropathy by activation of the p38MAPK/mTOR/ULK1 ser757 signaling pathway-Reference-Cited by-同舟云学术

sPLA2‐IB and PLA2R mediate insufficient autophagy and contribute to podocyte injury in idiopathic membranous nephropathy by activation of the p38MAPK/mTOR/ULK1 ser757 signaling pathway

Published:2020-11-13 Issue:2 Volume:35 Page:
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Yang Liyan¹,Wu Yuansheng²,Lin Songhua¹,Dai Binbin¹,Chen Hong³,Tao Xuan³,Li Guoping³,Wan Jianxin¹^ORCID,Pan Yangbin¹

Affiliation:

1. Department of Nephrology the First Affiliated Hospital of Fujian Medical University Fuzhou China

2. Department of Cardiology the First Affiliated Hospital of Fujian Medical University Fuzhou China

3. Department of Pathology the First Affiliated Hospital of Fujian Medical University Fuzhou China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.202001143R

Reference53 articles.

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4. Immune Response against Autoantigen PLA2R Is not Gambling: Implications for Pathophysiology, Prognosis, and Therapy

5. Serum secretory phospholipase A2 group IB correlates with the severity of membranous nephropathy

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