Advanced glycation end-products suppress autophagic flux in podocytes by activating mammalian target of rapamycin and inhibiting nuclear translocation of transcription factor EB-Reference-Cited by-同舟云学术

Advanced glycation end-products suppress autophagic flux in podocytes by activating mammalian target of rapamycin and inhibiting nuclear translocation of transcription factor EB

Published:2018-04-30 Issue:2 Volume:245 Page:235-248
ISSN:0022-3417
Container-title:The Journal of Pathology
language:en
Short-container-title:J. Pathol

Author:

Zhao Xingchen¹²^ORCID,Chen Yuanhan¹²,Tan Xiaofan²³,Zhang Li²,Zhang Hong²,Li Zhilian²,Liu Shuangxin²,Li Ruizhao²,Lin Ting²,Liao Ruyi²,Zhang Qianmei²,Dong Wei²,Shi Wei¹²,Liang Xinling²

Affiliation:

1. The Second School of Clinical Medicine; Southern Medical University; Guangzhou Guangdong PR China

2. Division of Nephrology, Guangdong General Hospital; Guangdong Academy of Medical Sciences, Guangdong Provincial Institute of Geriatrics; Guangzhou Guangdong PR China

3. Division of Nephrology; Zhongshan City People's Hospital, Zhongshan Hospital of Sun Yat-sen University; Zhongshan Guangdong PR China

Funder

Natural Science Foundation of Guangdong Province

National Clinical Key Specialty Construction Projects

National Natural Science Foundation of China

Publisher

Wiley

Subject

Pathology and Forensic Medicine

Reference44 articles.

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2. podocytes lose their footing;Rask-Madsen;Nature,2010