Over-expression of extracellular superoxide dismutase in mouse synovial tissue attenuates the inflammatory arthritis-Reference-Cited by-同舟云学术

Over-expression of extracellular superoxide dismutase in mouse synovial tissue attenuates the inflammatory arthritis

Published:2012 Issue:9 Volume:44 Page:529
ISSN:1226-3613
Container-title:Experimental & Molecular Medicine
language:en
Short-container-title:Exp Mol Med

Author:

Yu Dong Hoon¹,Yi Jun Koo¹,Yuh Hyung Soo¹,Park Seo jin¹,Kim Hei Jung¹,Bae Ki Beom²,Ji Young Rae¹,Kim Na Ri¹,Park Si Jun¹,Kim Do Hyung³,Kim Sung Hyun¹,Kim Myoung Ok⁴,Lee Jeong Woong⁵,Ryoo Zae Young¹

Affiliation:

1. School of Life Sciences and Biotechnology, Kyungpook National University, Daegu 702-701, Korea.

2. Hormel Institute, University of Minnesota, Minnesota 55912, USA.

3. Department of Physics, Kyungpook National University, Daegu 702-701, Korea.

4. Department of BT, Kyungpook National University, Sangju 742-711, Korea.

5. Regenerative Medicine Research Center, Korea Research Institute of Bioscience and Biotechnology, Daejeon 305-806, Korea.

Publisher

Springer Science and Business Media LLC

Subject

Clinical Biochemistry,Molecular Biology,Molecular Medicine,Biochemistry

Reference33 articles.

1. Nox4 as the Major Catalytic Component of an Endothelial NAD(P)H Oxidase

2. Cell contact between T cells and synovial fibroblasts causes induction of adhesion molecules and cytokines

3. Oxygen radicals as effectors of cartilage destruction. Direct degradative effect on matrix components and indirect action via activation of latent collagenase from polymorphonuclear leukocytes

4. Mice lacking extracellular superoxide dismutase are more sensitive to hyperoxia.

5. Superoxide production and NADPH oxidase expression in human rheumatoid synovial cells: regulation by interleukin-1β and tumour necrosis factor-α

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