Differential Transcriptional Responses to Interferon-α and Interferon-γ in Primary Human Hepatocytes
Author:
Affiliation:
1. Department of Medicine, Stanford University School of Medicine, Stanford, California.
2. VA Palo Alto Health Care System, Palo Alto, California.
3. Liver Diseases Branch, NIDDK, NIH, Bethesda, Maryland.
Publisher
Mary Ann Liebert Inc
Subject
Virology,Cell Biology,Immunology
Link
http://www.liebertpub.com/doi/pdf/10.1089/jir.2009.0082
Reference40 articles.
1. Replication of Hepatitis C Virus (HCV) RNA in Mouse Embryonic Fibroblasts: Protein Kinase R (PKR)-Dependent and PKR-Independent Mechanisms for Controlling HCV RNA Replication and Mediating Interferon Activities
2. Identification of genes differentially regulated by interferon , , or using oligonucleotide arrays
3. Hepatic gene expression during treatment with peginterferon and ribavirin: Identifying molecular pathways for treatment response
4. Interferon-γ inhibits replication of subgenomic and genomic hepatitis C virus RNAs
5. Peginterferon Alfa-2a plus Ribavirin for Chronic Hepatitis C Virus Infection
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