NF-κB/Cartilage Acidic Protein 1 Promotes Ultraviolet B Irradiation-Induced Apoptosis of Human Lens Epithelial Cells-Reference-Cited by-同舟云学术

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NF-κB/Cartilage Acidic Protein 1 Promotes Ultraviolet B Irradiation-Induced Apoptosis of Human Lens Epithelial Cells

Published:2020-04-01 Issue:4 Volume:39 Page:513-521
ISSN:1044-5498
Container-title:DNA and Cell Biology
language:en
Short-container-title:DNA and Cell Biology

Author:

Sun Yang¹²³⁴⁵,Rong Xianfang¹²³⁴⁵,Li Dan¹²³⁴⁵,Lu Yi¹²³⁴⁵,Ji Yinghong¹²³⁴⁵

Affiliation:

1. Department of Ophthalmology, Eye & ENT Hospital of Fudan University, Shanghai, China.

2. Eye Institute, Eye & ENT Hospital of Fudan University, Shanghai, China.

3. National Health Commission (NHC) Key Laboratory of Myopia (Fudan University), Shanghai, China.

4. Laboratory of Myopia, Chinese Academy of Medical Sciences, Shanghai, China.

5. Key Laboratory of Visual Impairment and Restoration of Shanghai, Shanghai, China.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Genetics,Molecular Biology,General Medicine

Link

https://www.liebertpub.com/doi/pdf/10.1089/dna.2019.5086

Reference33 articles.

1. Histidine protects human lens epithelial cells against H 2 O 2 ‐induced oxidative stress injury through the NF‐кB pathway

2. Inhibition of p38 mitogen-activated protein kinase phosphorylation decreases H2O2-induced apoptosis in human lens epithelial cells

3. Failure to activate NF-κB promotes apoptosis of retinal ganglion cells following optic nerve transection

4. Mechanisms and functions of p38 MAPK signalling

5. H2O2-mediated oxidative stress activates NF-κB in lens epithelial cells

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