Failure to activate NF-κB promotes apoptosis of retinal ganglion cells following optic nerve transection-Reference-Cited by-同舟云学术

Failure to activate NF-κB promotes apoptosis of retinal ganglion cells following optic nerve transection

Published:2000-11 Issue:1 Volume:883 Page:60-68
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Choi Jun-Sub,Kim Jeong-a.,Kim Dong-Hwan,Chun Myung-Hun,Gwag Byung J.,Yoon Sungjoo Kim,Joo Choun-Ki

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference43 articles.

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3. Drug-induced neuroprotection from global ischemia is associated with prevention of persistent but not transient activation of nuclear factor-κB in rat;Clemens;Stroke,1998

4. Salicylates and sufaalazine, but not glucocorticoids, inhibit leukocyte accumulation by an adenosine-dependent mechanism that is independent of inhibition of prostaglandin synthesis and p105 of NF-κB;Cronstein;Proc. Natl. Acad. Sci. USA,1999

5. CNTF, not othertrophic factors, promotes axonal regeneration of axotomized retinal ganglion cells in adult hamsters;Cui;Invest. Ophthalmol. Vis. Sci.,1999

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