Frataxin Deficiency Leads to Defects in Expression of Antioxidants and Nrf2 Expression in Dorsal Root Ganglia of the Friedreich's Ataxia YG8R Mouse Model-Reference-Cited by-同舟云学术

Frataxin Deficiency Leads to Defects in Expression of Antioxidants and Nrf2 Expression in Dorsal Root Ganglia of the Friedreich's Ataxia YG8R Mouse Model

Published:2013-11 Issue:13 Volume:19 Page:1481-1493
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Shan Yuxi¹,Schoenfeld Robert A.¹,Hayashi Genki¹,Napoli Eleonora¹,Akiyama Tasuku²,Iodi Carstens Mirela²,Carstens Earl E.²,Pook Mark A.³,Cortopassi Gino A.¹

Affiliation:

1. Department of Molecular Biosciences, University of California, Davis, California.

2. Department of Neurobiology, Physiology and Behavior, University of California, Davis, California.

3. Biosciences, School of Health Sciences & Social Care, Brunel University, Uxbridge, United Kingdom.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2012.4537

Reference81 articles.

1. Distal Airways in Mice Exposed to Cigarette Smoke

2. GAA repeat instability in Friedreich ataxia YAC transgenic mice

3. GAA repeat expansion mutation mouse models of Friedreich ataxia exhibit oxidative stress leading to progressive neuronal and cardiac pathology

4. RNAi-mediated suppression of the mitochondrial iron chaperone, frataxin, in Drosophila

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