Deciphering the ferroptosis pathways in dorsal root ganglia of Friedreich ataxia models. The role of LKB1/AMPK, KEAP1, and GSK3β in the impairment of the NRF2 response-Reference-Cited by-同舟云学术

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Deciphering the ferroptosis pathways in dorsal root ganglia of Friedreich ataxia models. The role of LKB1/AMPK, KEAP1, and GSK3β in the impairment of the NRF2 response

Published:2024-10 Issue: Volume:76 Page:103339
ISSN:2213-2317
Container-title:Redox Biology
language:en
Short-container-title:Redox Biology

Author:

Sanz-Alcázar Arabela^ORCID,Portillo-Carrasquer Marta^ORCID,Delaspre Fabien^ORCID,Pazos-Gil Maria^ORCID,Tamarit Jordi^ORCID,Ros Joaquim^ORCID,Cabiscol Elisa^ORCID

Funder

Government of Catalonia

Universitat de Lleida

Ministerio de Economía y Competitividad

Generalitat de Catalunya Departament d'Innovacio Universitats I Empresa

Publisher

Elsevier BV

Reference72 articles.

1. Friedreich's ataxia: autosomal recessive disease caused by an lntronic GAA triplet repeat expansion;Campuzano;Science,1996

2. Compound heterozygous FXN mutations and clinical outcome in friedreich ataxia;Galea;Ann. Neurol.,2016

3. Clinical features of Friedreich's ataxia: classical and atypical phenotypes;Parkinson;J. Neurochem.,2013

4. Mortality in friedreich ataxia;Tsou;J. Neurol. Sci.,2011

5. Clinical and genetic abnormalities in patients with Friedreich's ataxia;Dürr;N. Engl. J. Med.,1996

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