Mitochondrial Dysfunction in Aging and Longevity: A Causal or Protective Role?-Reference-Cited by-同舟云学术

Mitochondrial Dysfunction in Aging and Longevity: A Causal or Protective Role?

Published:2013-10-20 Issue:12 Volume:19 Page:1373-1387
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Pulliam Daniel A.¹²,Bhattacharya Arunabh¹²,Van Remmen Holly¹²³

Affiliation:

1. Barshop Institute for Longevity and Aging Studies, University of Texas Health Science Center at San Antonio, San Antonio, Texas.

2. Department of Cellular and Structural Biology, San Antonio, Texas.

3. GRECC, South Texas Veterans Health Care System, San Antonio, Texas.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2012.4950

Reference97 articles.

1. Effects of aging on respiration, ATP levels and calcium transport in rat liver mitochondria. Response to theophylline

2. Initial evidence linking synaptic superoxide production with poor short-term memory in aged mice

3. Aging and acute exercise enhance free radical generation in rat skeletal muscle

4. Long-term caloric restriction increases UCP3 content but decreases proton leak and reactive oxygen species production in rat skeletal muscle mitochondria

5. Uncoupling the Pleiotropic Phenotypes of clk-1 with tRNA Missense Suppressors in Caenorhabditis elegans

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