Mutyh deficiency downregulates mitochondrial fusion proteins and causes cardiac dysfunction via α-ketoglutaric acid reduction with oxidative stress-Reference-Cited by-同舟云学术

Mutyh deficiency downregulates mitochondrial fusion proteins and causes cardiac dysfunction via α-ketoglutaric acid reduction with oxidative stress

Published:2022-02-01 Issue:2 Volume:56 Page:129-142
ISSN:1071-5762
Container-title:Free Radical Research
language:en
Short-container-title:Free Radical Research

Author:

Chen Jingwen¹²,Wu Xin¹²,Wang Yanyi¹²,Pan Yunfeng¹²,Ren Yan¹²,Nakabeppu Yusaku³,Fan Yimei¹²,Wang Yaping¹²

Affiliation:

1. Department of Medical Genetics, Nanjing University School of Medicine, Nanjing, China

2. Jiangsu Key Laboratory of Molecular Medicine, Nanjing University, Nanjing, China

3. Division of Neurofunctional Genomics, Department of Immunobiology and Neuroscience, Medical Institute of Bioregulation, Kyushu University, Fukuoka, Japan

Funder

National Natural Science Foundation of China

Publisher

Informa UK Limited

Subject

General Medicine,Biochemistry

Link

https://www.tandfonline.com/doi/pdf/10.1080/10715762.2022.2036336

Reference48 articles.

1. Functional Expression of hMYH, a Human Homolog of the Escherichia coli MutY Protein

2. Oxidative DNA damage & repair: An introduction

3. Inherited variants of MYH associated with somatic G:C→T:A mutations in colorectal tumors

4. AluYb8 insertion in the MUTYH gene is related to increased 8-OHdG in genomic DNA and could be a risk factor for type 2 diabetes in a Chinese population

5. AluYb8 insertion polymorphism in the MUTYH gene impairs mitochondrial DNA maintenance and affects the age of onset of IPF

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