Activated PI3K/Akt/COX-2 Pathway Induces Resistance to Radiation in Human Cervical Cancer HeLa Cells-Reference-Cited by-同舟云学术

Activated PI3K/Akt/COX-2 Pathway Induces Resistance to Radiation in Human Cervical Cancer HeLa Cells

Published:2010-06 Issue:3 Volume:25 Page:317-323
ISSN:1084-9785
Container-title:Cancer Biotherapy and Radiopharmaceuticals
language:en
Short-container-title:Cancer Biotherapy and Radiopharmaceuticals

Author:

Xia Shu¹,Zhao Yin²,Yu Shiying¹,Zhang Mengxian¹

Affiliation:

1. Department of Oncology, The Tong Ji Hospital, TongJi Medical College, HuaZhong University of Science and Technology, Wu Han, China.

2. Department of Obstetrics and Gynecology, The Union Hospital, TongJi Medical College, HuaZhong University of Science and Technology, Wu Han, China.

Publisher

Mary Ann Liebert Inc

Subject

Cancer Research,Pharmacology,Radiology, Nuclear Medicine and imaging,Oncology,General Medicine,Cancer Research,Pharmacology,Radiology, Nuclear Medicine and imaging,Oncology,General Medicine

Link

http://www.liebertpub.com/doi/pdf/10.1089/cbr.2009.0707

Reference44 articles.

1. Akt inhibits Myt1 in the signalling pathway that leads to meiotic G2/M-phase transition

2. Activation of Akt/Protein Kinase B Overcomes a G 2 /M Cell Cycle Checkpoint Induced by DNA Damage

3. Phosphatidylinositol 3-Kinase Inhibition Broadly Sensitizes Glioblastoma Cells to Death Receptor– and Drug-Induced Apoptosis

4. Assigning Functional Domains within the p101 Regulatory Subunit of Phosphoinositide 3-Kinase γ

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