Liver Sinusoidal Endothelial Cells at the Crossroad of Iron Overload and Liver Fibrosis
Author:
Affiliation:
1. Department of Molecular Biotechnology and Health Sciences, Molecular Biotechnology Center, University of Torino, Torino, Italy.
2. Institute of Biostructure and Bioimaging, CNR c/o Molecular Biotechnology Center, Torino, Italy.
Publisher
Mary Ann Liebert Inc
Subject
Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry
Link
https://www.liebertpub.com/doi/pdf/10.1089/ars.2020.8168
Reference125 articles.
1. Dietary iron restriction leads to a reduction in hepatic fibrosis in a rat model of non-alcoholic steatohepatitis
2. Hepatic nerve growth factor induced by iron overload triggers defenestration in liver sinusoidal endothelial cells
3. SLN124, a GalNAc-siRNA Conjugate Targeting TMPRSS6, Efficiently Prevents Iron Overload in Hereditary Haemochromatosis Type 1
4. BMP6 is a key endogenous regulator of hepcidin expression and iron metabolism
5. Burden of liver diseases in the world
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1. Secondary iron overload induces chronic pancreatitis and ferroptosis of acinar cells in mice;International Journal of Molecular Medicine;2022-12-09
2. Liver Iron Loading in Alcohol-Associated Liver Disease;The American Journal of Pathology;2022-10
3. Liver Fibrosis Assessment by Viewing Sinusoidal Capillarization: US Molecular Imaging versus Two-dimensional Shear-Wave Elastography in Rats;Radiology;2022-08
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