YB-1 Recruits Drosha to Promote Splicing of <i>pri-miR-192</i> to Mediate the Proangiogenic Effects of H<sub>2</sub>S-Reference-Cited by-同舟云学术

YB-1 Recruits Drosha to Promote Splicing of pri-miR-192 to Mediate the Proangiogenic Effects of H₂S

Published:2022-04-01 Issue:10-12 Volume:36 Page:760-783
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Zhou Yu¹,Li Xing-Hui¹²,Xue Wen-Long¹,Jin Sheng¹³,Li Meng-Yao¹,Zhang Cai-Cai¹⁴,Yu Bo⁵,Zhu Lei⁵,Liang Kun⁵,Chen Ying¹,Tao Bei-Bei¹,Zhu Yi-Zhun⁶⁷,Wang Ming-Jie¹,Zhu Yi-Chun¹^ORCID

Affiliation:

1. Shanghai Key Laboratory of Bioactive Small Molecules, Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Fudan University Shanghai Medical College, Shanghai, China.

2. Shanghai Cao Yang NO.2 High School, Shanghai, China.

3. Department of Physiology, Hebei Medical University, Shijiazhuang, China.

4. Department of Physiology, Hainan Medical College, Haikou, China.

5. Department of Vascular Surgery, Huashan Hospital, Fudan University, Shanghai, China.

6. Department of Pharmacology, School of Pharmacy, Fudan University, Shanghai, China.

7. State Key Laboratory of Quality Research in Chinese Medicine and School of Pharmacy, Macau University of Science and Technology, Avenida WaiLong, Taipa, China.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

https://www.liebertpub.com/doi/pdf/10.1089/ars.2021.0105

Reference50 articles.

1. Calcineurin-mediated YB-1 Dephosphorylation Regulates CCL5 Expression during Monocyte Differentiation

2. Contact inhibition modulates intracellular levels of miR-223 in a p27kip1-dependent manner

3. Inhibition of Protein Synthesis by Y Box-Binding Protein 1 Blocks Oncogenic Cell Transformation

4. The novel proangiogenic effect of hydrogen sulfide is dependent on Akt phosphorylation

5. Acetylation of p53 stimulates miRNA processing and determines cell survival following genotoxic stress

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