Acetylation of p53 stimulates miRNA processing and determines cell survival following genotoxic stress-Reference-Cited by-同舟云学术

Acetylation of p53 stimulates miRNA processing and determines cell survival following genotoxic stress

Published:2013-11-12 Issue:24 Volume:32 Page:3192-3205
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:

Author:

Chang Jonathan¹²³,Davis-Dusenbery Brandi N²,Kashima Risa¹,Jiang Xuan¹,Marathe Nisha¹,Sessa Roberto¹,Louie Justin¹,Gu Wei⁴,Lagna Giorgio¹²⁵,Hata Akiko¹²³

Affiliation:

1. Cardiovascular Research Institute, University of California; San Francisco CA USA

2. Molecular Cardiology Research Institute, Tufts Medical Center; Boston MA USA

3. Department of Biochemistry, Tufts University School of Medicine; Boston MA USA

4. Department of Pathology and Cell Biology, Institute of Cancer Genetics, College of Physicians and Surgeons, Columbia University; New York NY USA

5. Department of Cellular and Molecular Pharmacology, University of California; San Francisco CA USA

Publisher

Wiley

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Reference38 articles.

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2. microRNA-203 suppresses bladder cancer development by repressing bcl-2 expression;Bo;FEBS J,2011

3. The impact of acetylation and deacetylation on the p53 pathway;Brooks;Protein Cell,2011

4. Genetic and epigenetic silencing of microRNA-203 enhances ABL1 and BCR-ABL1 oncogene expression;Bueno;Cancer Cell,2008

5. Requirement for p53 and p21 to sustain G2 arrest after DNA damage;Bunz;Science,1998

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