Efficiency of CCR5 Coreceptor Utilization by the HIV Quasispecies Increases over Time, But Is Not Associated with Disease Progression-Reference-Cited by-同舟云学术

Efficiency of CCR5 Coreceptor Utilization by the HIV Quasispecies Increases over Time, But Is Not Associated with Disease Progression

Published:2012-03 Issue:3 Volume:28 Page:289-294
ISSN:0889-2229
Container-title:AIDS Research and Human Retroviruses
language:en
Short-container-title:AIDS Research and Human Retroviruses

Author:

Redd Andrew D.¹,Laeyendecker Oliver¹²,Kong Xiangrong³,Kiwanuka Noah⁴⁵⁶,Lutalo Tom⁴,Huang Wei⁷,Gray Ronald H.³,Wawer Maria J.³,Serwadda David⁶,Eshleman Susan H.²,Quinn, on behalf of the Rakai Healt Thomas C.¹²

Affiliation:

1. Laboratory of Immunoregulation, NIAID, NIH, Bethesda, Maryland.

2. School of Medicine, Johns Hopkins University, Baltimore, Maryland.

3. Bloomberg School of Public Health, Johns Hopkins University, Baltimore, Maryland.

4. Rakai Health Sciences Program, Rakai District, Uganda.

5. Uganda Virus Research Institute, Entebbe, Uganda.

6. School of Public Health, Makerere University, Kampala, Uganda.

7. Monogram Biosciences Inc., South San Francisco, California.

Publisher

Mary Ann Liebert Inc

Subject

Infectious Diseases,Virology,Immunology

Link

http://www.liebertpub.com/doi/pdf/10.1089/aid.2011.0006

Reference25 articles.

1. The HIV-1 Envelope Glycoproteins: Fusogens, Antigens, and Immunogens

2. Virus Entry via the Alternative Coreceptors CCR3 and FPRL1 Differs by Human Immunodeficiency Virus Type 1 Subtype

3. Genetic Restriction of HIV-1 Infection and Progression to AIDS by a Deletion Allele of the CKR5 Structural Gene

4. Enhanced CD4+ cellular apoptosis by CCR5-restricted HIV-1 envelope glycoprotein variants from patients with progressive HIV-1 infection

5. Killing of primary CD4+ T cells by non-syncytium-inducing macrophage-tropic human immunodeficiency virus type 1.

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