New Insights into the Role of Histone Deacetylases as Coactivators of Inflammatory Gene Expression-Reference-Cited by-同舟云学术

New Insights into the Role of Histone Deacetylases as Coactivators of Inflammatory Gene Expression

Published:2015-07 Issue:1 Volume:23 Page:85-98
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Schmitz Michael Lienhard¹²,de la Vega Laureano³

Affiliation:

1. Medical Faculty, Institute of Biochemistry, Justus-Liebig-University, Giessen, Germany.

2. The German Center for Lung Research, Giessen, Germany.

3. Division of Cancer Research, Medical Research Institute, Jacqui Wood Cancer Centre, University of Dundee, Ninewells Hospital and Medical School, Dundee, United Kingdom.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2013.5750

Reference145 articles.

1. Tandem PHD Fingers of MORF/MOZ Acetyltransferases Display Selectivity for Acetylated Histone H3 and Are Required for the Association with Chromatin

2. STAT1 Signaling Is Not Regulated by a Phosphorylation-Acetylation Switch

3. Purely stochastic binary decisions in cell signaling models without underlying deterministic bistabilities

4. The p65 (RelA) Subunit of NF-κB Interacts with the Histone Deacetylase (HDAC) Corepressors HDAC1 and HDAC2 To Negatively Regulate Gene Expression

5. Tumor necrosis factor and interleukin-1 lead to phosphorylation and loss of I kappa B alpha: a mechanism for NF-kappa B activation.

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