Lipidomic Analysis of Apoptotic Hela Cells Induced by Paclitaxel
Author:
Affiliation:
1. Key Laboratory of Systems Bioengineering, Ministry of Education and Department of Pharmaceutical Engineering, School of Chemical Engineering and Technology, Tianjin University, Tianjin, People's Republic of China.
Publisher
Mary Ann Liebert Inc
Subject
Genetics,Molecular Biology,Molecular Medicine,Biochemistry,Biotechnology
Link
http://www.liebertpub.com/doi/pdf/10.1089/omi.2011.0027
Reference43 articles.
1. Inhibition of Phosphatidylcholine Biosynthesis following Induction of Apoptosis in HL-60 Cells
2. Cell membranes as target for anticancer agents
3. Reversal of Tumor Resistance to Apoptotic Stimuli by Alteration of Membrane Fluidity: Therapeutic Implications
4. Akt is upstream and MAPKs are downstream of NF-κB in paclitaxel-induced survival signaling events, which are down-regulated by curcumin contributing to their synergism
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