Gap-Junction Channels Dysfunction in Deafness and Hearing Loss-Reference-Cited by-同舟云学术

Gap-Junction Channels Dysfunction in Deafness and Hearing Loss

Published:2009-02 Issue:2 Volume:11 Page:309-322
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Martínez Agustín D.¹,Acuña Rodrigo¹,Figueroa Vania¹,Maripillan Jaime¹,Nicholson Bruce²

Affiliation:

1. Centro de Neurociencias de Valparaíso, Universidad de Valparaíso. Valparaíso, Chile.

2. Department of Biochemistry, The University of Texas Health Science Center, San Antonio. Texas.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2008.2138

Reference114 articles.

1. Connexins 26 and 30 are co-assembled to form gap junctions in the cochlea of mice

2. A Novel N14Y Mutation in Connexin26 in Keratitis-Ichthyosis-Deafness Syndrome

3. Targeted epidermal expression of mutant Connexin 26(D66H) mimics true Vohwinkel syndrome and provides a model for the pathogenesis of dominant connexin disorders

4. Impaired permeability to Ins(1,4,5)P3 in a mutant connexin underlies recessive hereditary deafness

5. New roles for astrocytes: Gap junction hemichannels have something to communicate

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