Superoxide is an Intrinsic Signaling Molecule Triggering Muscle Hypertrophy-Reference-Cited by-同舟云学术

Superoxide is an Intrinsic Signaling Molecule Triggering Muscle Hypertrophy

Published:2024-07-12 Issue: Volume: Page:
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Lu Siyu¹,Zhou Yiming¹,Liu Mincong¹,Gong Lijun¹,Liu Li¹,Duan Zhigui¹,Chen Keke¹,Gonzalez Frank J.²,Wei Fang¹³⁴,Xiang Rong⁵,Li Guolin¹³⁴^ORCID

Affiliation:

1. Center for Aging Biomedicine, National & Local Joint Engineering Laboratory of Animal Peptide Drug Development, College of Life Sciences, Hunan Normal University, Changsha, China.

2. Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, USA.

3. Key Laboratory of Hunan Province for Model Animal and Stem Cell Biology, School of Medicine, Hunan Normal University, Changsha, China.

4. FuRong Laboratory, Changsha, China.

5. Department of Cell Biology, School of Life Sciences, Central South University, Changsha, China.

Publisher

Mary Ann Liebert Inc

Link

https://www.liebertpub.com/doi/pdf/10.1089/ars.2024.0595

Reference56 articles.

1. A novel flow cytometry assay using dihydroethidium as redox-sensitive probe reveals NADPH oxidase-dependent generation of superoxide anion in human platelets exposed to amyloid peptide β

2. Neocuproine, a selective Cu(I) chelator, and the relaxation of rat vascular smooth muscle by S -nitrosothiols

3. Tetrathiomolybdate Inhibits Copper Trafficking Proteins Through Metal Cluster Formation

4. Molecular basis for muscle loss that causes cachexia

5. Carnosine and neocuproine as neutralizing agents for copper overload-induced damages in cultured human cells

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