Britanin Ameliorates Cerebral Ischemia–Reperfusion Injury by Inducing the Nrf2 Protective Pathway-Reference-Cited by-同舟云学术

Britanin Ameliorates Cerebral Ischemia–Reperfusion Injury by Inducing the Nrf2 Protective Pathway

Published:2017-10-10 Issue:11 Volume:27 Page:754-768
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Wu Guozhen¹,Zhu Lili²,Yuan Xing¹,Chen Hao¹,Xiong Rui²,Zhang Shoude²,Cheng Hao³,Shen Yunheng¹,An Huazhang⁴,Li Tiejun³,Li Honglin²,Zhang Weidong¹

Affiliation:

1. Department of Phytochemistry, School of Pharmacy, Second Military Medical University, Shanghai, P.R. China.

2. State Key Laboratory of Bioreactor Engineering, Shanghai Key Laboratory of New Drug Design, School of Pharmacy, East China University of Science and Technology, Shanghai, P.R. China.

3. Department of Pharmacology, School of Pharmacy, Second Military Medical University, Shanghai, P.R. China.

4. Cancer Institute, Institute of Translational Medicine, Second Military Medical University, Shanghai, P.R. China.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2016.6885

Reference43 articles.

1. Sulforaphane preconditioning of the Nrf2/HO-1 defense pathway protects the cerebral vasculature against blood–brain barrier disruption and neurological deficits in stroke

2. Phosphorylation of Nrf2 in the transcription activation domain by casein kinase 2 (CK2) is critical for the nuclear translocation and transcription activation function of Nrf2 in IMR-32 neuroblastoma cells

3. iMOSFLM: a new graphical interface for diffraction-image processing withMOSFLM

4. Dimethyl fumarate in the treatment of relapsing–remitting multiple sclerosis: an overview

5. Oxidative Stress in Ischemic Brain Damage: Mechanisms of Cell Death and Potential Molecular Targets for Neuroprotection

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