Oxidative Stress in Ischemic Brain Damage: Mechanisms of Cell Death and Potential Molecular Targets for Neuroprotection-Reference-Cited by-同舟云学术

Oxidative Stress in Ischemic Brain Damage: Mechanisms of Cell Death and Potential Molecular Targets for Neuroprotection

Published:2011-04-15 Issue:8 Volume:14 Page:1505-1517
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Chen Hai¹,Yoshioka Hideyuki¹,Kim Gab Seok¹,Jung Joo Eun¹,Okami Nobuya¹,Sakata Hiroyuki¹,Maier Carolina M.¹,Narasimhan Purnima¹,Goeders Christina E.¹,Chan Pak H.¹

Affiliation:

1. Departments of Neurosurgery and Neurology and Neurological Sciences and Program in Neurosciences, Stanford University School of Medicine, Stanford, California.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2010.3576

Reference79 articles.

1. Three Distinct Mechanisms Generate Oxygen Free Radicals in Neurons and Contribute to Cell Death during Anoxia and Reoxygenation

2. Phospholipase A2, Hydroxyl Radicals, and Lipid Peroxidation in Transient Cerebral Ischemia

3. Effects of Matrix Metalloproteinase-9 Gene Knock-Out on the Proteolysis of Blood–Brain Barrier and White Matter Components after Cerebral Ischemia

4. Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide.

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