MicroRNA-21 Mediates Angiotensin II-Induced Liver Fibrosis by Activating NLRP3 Inflammasome/IL-1β Axis via Targeting Smad7 and Spry1-Reference-Cited by-同舟云学术

MicroRNA-21 Mediates Angiotensin II-Induced Liver Fibrosis by Activating NLRP3 Inflammasome/IL-1β Axis via Targeting Smad7 and Spry1

Published:2017-07 Issue:1 Volume:27 Page:1-20
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Ning Zuo-Wei¹,Luo Xiao-Ying¹,Wang Guo-Zhen¹,Li Yang¹,Pan Miao-Xia²,Yang Ren-Qiang³,Ling Xu-Guang³,Huang Shan¹,Ma Xiao-Xin¹,Jin Si-Yi¹,Wang Dan¹,Li Xu¹⁴

Affiliation:

1. Guangdong Provincial Key Laboratory of Gastroenterology, Department of Gastroenterology, Nanfang Hospital, Southern Medical University, Guangzhou, China.

2. Department of Respiratory Diseases, Southern Medical University, Guangzhou, China.

3. Department of Emergency Medicine, Nanfang Hospital, Southern Medical University, Guangzhou, China.

4. State Key Laboratory of Organ Failure Research, Guangdong Provincial Key Laboratory of Viral Hepatitis Research, Department of Infectious Diseases, Nanfang Hospital, Southern Medical University, Guangzhou, China.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2016.6669

Reference50 articles.

1. Restoration of muscle strength in dystrophic muscle by angiotensin-1-7 through inhibition of TGF-β signalling

2. Role of miR-21 in the pathogenesis of atrial fibrosis

3. The NOX toolbox: validating the role of NADPH oxidases in physiology and disease

4. Angiotensin-(1–7) Improves Liver Fibrosis by Regulating the NLRP3 Inflammasome via Redox Balance Modulation

5. Role of Nox isoforms in angiotensin II-induced oxidative stress and endothelial dysfunction in brain

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