Glutaredoxin1 Diminishes Amyloid Beta-Mediated Oxidation of F-Actin and Reverses Cognitive Deficits in an Alzheimer's Disease Mouse Model-Reference-Cited by-同舟云学术

Glutaredoxin1 Diminishes Amyloid Beta-Mediated Oxidation of F-Actin and Reverses Cognitive Deficits in an Alzheimer's Disease Mouse Model

Published:2019-12-20 Issue:18 Volume:31 Page:1321-1338
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Kommaddi Reddy Peera¹,Tomar Deepika Singh¹,Karunakaran Smitha¹,Bapat Deepti¹,Nanguneri Siddharth¹,Ray Ajit¹,Schneider Bernard L.²,Nair Deepak¹,Ravindranath Vijayalakshmi¹³

Affiliation:

1. Centre for Neuroscience, Indian Institute of Science, Bangalore, India.

2. Brain Mind Institute, Ecole Polytechnique Fédérale de Lausanne, Lausanne, Switzerland.

3. Centre for Brain Research, Indian Institute of Science, Bangalore, India.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

https://www.liebertpub.com/doi/pdf/10.1089/ars.2019.7754

Reference70 articles.

1. Reactive Oxygen Species-Mediated Loss of Synaptic Akt1 Signaling Leads to Deficient Activity-Dependent Protein Translation Early in Alzheimer's Disease

2. Protein oxidation in the brain in Alzheimer's disease

3. Changes in thiol content and expression of glutathione redox system genes in the hippocampus and cerebellum in Alzheimer's disease

4. Involvement of glutaredoxin-1 and thioredoxin-1 in β-amyloid toxicity and Alzheimer's disease

5. Oxidative Stress in the Progression of Alzheimer Disease in the Frontal Cortex

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